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Therefore, this review summarizes the mechanisms of ferroptosis, its functional impacts on wound repair-related cells (macrophages, fibroblasts, endothelial cells, keratinocytes) and wound bacterial infections

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Experiments with retinal endothelial cells showed, in turn, that their exposure to carboplatin results in an increased production of ICAM-1 and IL-8, which was followed by their increased proliferation, migration and apoptosis [74]

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