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In contrast, clinical metabolomic analyses of KD have reported increases in specific BCAAs, such as valine, and improvement of AD-related risk factors such as increased HDL-C and reduced BMI, suggesting that BCAA dynamics may differ depending on the disease stage and metabolic context [35]

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On top of understanding the microbial pathogenesis and the molecular and cellular mechanisms of disease biology, activity of a therapeutic agent in a translational research, be it either in vitro or in vivo, is critical to proffer advancement to first-in-human clinical trials based on laboratory findings of pharmacology, toxicology, and immunology

Interestingly, in a mouse model of turpentine-induced tissue inflammation, high D3 expression in invading granulocytes has also been reported (157, 158)